|本期目录/Table of Contents|

[1]朱玉芬综述,李崖青,付 丽审校.肿瘤相关巨噬细胞与乳腺癌关系的研究进展[J].天津医科大学学报,2014,20(06):499-501.
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肿瘤相关巨噬细胞与乳腺癌关系的研究进展(PDF)
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《天津医科大学学报》[ISSN:1006-8147/CN:12-1259/R]

卷:
20卷
期数:
2014年06期
页码:
499-501
栏目:
综述
出版日期:
2014-11-20

文章信息/Info

Title:
-
文章编号:
1006-8147(2014)06-0499-03
作者:
朱玉芬综述李崖青付 丽审校
(天津医科大学肿瘤医院乳腺病理研究室,天津市肿瘤防治重点实验室,乳腺癌防治教育部重点实验室,国家病理学重点学科,国家肿瘤临床医学研究中心,天津 300060)

Author(s):
-
关键词:
乳腺肿瘤肿瘤相关巨噬细胞肿瘤微环境
Keywords:
-
分类号:
R737.9
DOI:
-
文献标志码:
A
摘要:
-
Abstract:
-

参考文献/References:

[1]Tarin D. Clinical and biological implications of the tumor microenvironment[J]. Cancer Microenviron, 2012, 5(2): 95

[2]Tang X Q, Mo C F, Wang Y S, et al. Anti-tumour strategies aiming to target tumour-associated macrophages[J]. Immunology, 2013, 138(2): 93

[3]Lewis C E, Pollard J W. Distinct role of macrophages in different tumor microenvironments[J]. Cancer Res, 2006, 66(2): 605

[4]Biswas S K, Sica A, Lewis C E. Plasticity of macrophage function during tumor progression: Regulation by distinct molecular mechanisms[J]. J Immunol, 2008, 180(4): 2011

[5]Xu H X, Zhu J, Smith S, et al. Notch-RBP-J signaling regulates the transcription factor IRF8 to promote inflammatory macrophage polarization[J]. Nat Immunol, 2012, 13(7): 642

[6]Liu Q, Zheng J, Yin D D, et al. Monocyte to macrophage differentiation-associated (MMD) positively regulates ERK and Akt activation and TNF-alpha and NO production in macrophages[J]. Mol Biol Rep, 2012, 39(5): 5643

[7]Gyorki D E, Asselin-Labat M L, van Rooijen N, et al. Resident macrophages influence stem cell activity in the mammary gland[J]. Breast Cancer Res, 2009, 11(4): R62

[8]Jinushi M, Chiba S, Yoshiyama H A, et al. Tumor-associated macrophages regulate tumorigenicity and anticancer drug responses of Cancer stem/initiating cells[J]. Proc Natl Acad Sci U S A, 2011, 108(30): 12425

[9]Yang J, Liao D, Chen C, et al. Tumor-Associated macrophages regulate murine breast Cancer stem cells through a novel paracrine EGFR/Stat3/Sox-2 signaling pathway[J]. Stem Cells, 2013, 31(2): 248

[10]Patsialou A, Wyckoff J, Wang Ya RONG, et al. Invasion of human breast Cancer cells in vivo requires both paracrine and autocrine loops involving the Colony-Stimulating factor-1 receptor[J]. Cancer Res, 2009, 69(24): 9498

[11]Lu X, Kang Y  B. Chemokine (C-C motif) ligand 2 engages CCR2(+) stromal cells of monocytic origin to promote breast Cancer metastasis to lung and bone[J]. J Biol Chemi, 2009, 284(42): 29087

[12]Kristiansen M, Graversen J H, Jacobsen C, et al. Identification of the haemoglobin scavenger receptor[J]. Nature, 2001, 409(6817): 198

[13]Maniecki M B, Etzerodt A, Ulhoi B P, et al. Tumor-promoting macrophages induce the expression of the macrophage-specific receptor CD163 in malignant cells[J]. Int J Cancer, 2012, 131(10): 2320

[14]Galmbacher K, Heisig M, Hotz C A, et al. Shigella mediated depletion of macrophages in a murine breast Cancer model is associated with tumor regression[J]. PLoS One, 2010, 5(3): A93.

[15]Qian B Z, Pollard J W. Macrophage diversity enhances tumor progression and metastasis[J]. Cell, 2010, 141(1): 39

[16]Schoppmann S F, Fenzl A, Nagy K A, et al. VEGF-C expressing tumor-associated macrophages in lymphnode positive breast Cancer: impact on lymphangiogenesis and survival[J]. Surgery, 2006, 139(6): 839

[17]Vicioso L, Gonzalez F J, Alvarez M, et al. Elevated serum levels of vascular endothelial growth factor are associated with tumor-associated macrophages in primary breast cancer[J]. Am J Clin Pathol, 2006, 125(1): 111

[18]Levano K S, Jung E H, Kenny P A. Breast Cancer subtypes Express distinct receptor repertoires for tumor-associated macrophage derived cytokines[J]. Biochem Biophys Res Commun, 2011, 411(1): 107

[19]Prat A, Perou C M. Deconstructing the molecular portraits of breast cancer[J]. Mol Oncol, 2011, 5(1): 5

[20]Trusolino L, Bertotti A, Comoglio P M. Met signalling: principles and functions in development, organ regeneration and Cancer[J]. Nat Rev Mol Cell Biol, 2010, 11(12): 834

[21]Moorman A M, Vink R, Heijmans H J, et al. Theprognostic value of tumour-stroma ratio in triple-negative breast cancer[J]. Eur J Surg Oncol, 2012, 38(4): 307

[22]Campbell M J, Tonlaar N Y, Garwood E R, et al. Proliferating macrophages associated with high grade, hormone receptor negative breast cancer and poor clinical outcome[J]. Breast Cancer Res Treat, 2011, 128(3): 703

[23]Ch’ng E S,Jaafar H, Tuan sharif S E. Breast tumor angiogenesis and tumor-associated macrophages:histopathologist’s perspective[J]. Patholog Res Int , 2011: 572706

[24]Ch’ng E S. Tuan sharif SE,jaafar H.in human invasive breast ductal carcinoma,tumor stromal macrophages and tumor nest macrophages have distinct relationships with clinicopathological parameters and tumor angiogenesis[J]. Virchows Arch, 2013, 462(3): 257.

[25]Medrek C, Ponten F, Jirstrom K A. The presence of tumor associated macrophages in tumor stroma as a prognostic marker for breast Cancer patients[J]. BMC Cancer, 2012, 12 : 12

[26]Gottfried E, Kunz-Schughart L A, Weber A, et al. Expression of CD68 in non-myeloid cell types[J]. Scand J Immunol, 2008, 67(5): 453

[27]Mahmoud S , Lee A , Paish E C, et al. Tumour-infiltrating macrophages and clinical outcome in breast cancer[J]. J Clin Pathol, 2012, 65(2): 159

[28]Condeelis J, Pollard J W. Macrophages: obligate partners for tumor cell migration, invasion, and metastasis[J]. Cell, 2006, 124(2): 263

[29]Sinha P, Clements V K, Bunt S K, et al. Cross-talk between myeloid-derived suppressor cells and macrophages subverts tumor immunity toward a type 2 response[J]. J Immunol, 2007, 179(2): 977.

[30]Grugan K D, Mccabe F L, Kinder M, et al. Tumor-associated macrophages promote invasion while retaining Fc-Dependent anti-tumor function[J]. J Immunol, 2012, 189(11): 5457

[31]Huang Y H, Yuan J P, Righi E, et al. Vascular normalizing doses of antiangiogenic treatment reprogram the immunosuppressive tumor microenvironment and enhance immunotherapy[J]. Proc Natl Acad Sci U S A, 2012, 109(43): 17561

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[2]曹文庆,宁殿宾,冯 丽,等.氨肽酶N在乳腺癌血管生成中的作用及其与VEGF的相关性[J].天津医科大学学报,2015,21(01):39.
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[3]王月洁 综述,魏枫,任秀宝 审校.肿瘤相关巨噬细胞:守护者还是破坏者[J].天津医科大学学报,2021,27(02):199.
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备注/Memo

备注/Memo:
基金项目 国家自然科学基金重点项目资助(30930038)

作者简介 朱玉芬(1986 -),女,硕士在读,研究方向:肿瘤微环境与乳腺癌生长侵袭转移机制;通信作者:付丽,E-mail: fulijyb@hotmail.com

更新日期/Last Update: 2014-12-03