巨噬细胞在子宫内膜异位症发生发展中的作用研究现状-《天津医科大学学报》

参考文献/References:

[1] Miller J E, Ahn S H, Monsanto S P, et al. Implications of immune dysfunction on endometriosis associated infertility[J].Oncotarget,2017，8（4）:7138
[2] 刘颂平,温坚.子宫内膜异位症发病机制研究新进展[J]. 医学综述，2013，19（2）：291
[3] Vercellini P, Buggio L, Berlanda N, et al. Estrogen-progestins and progestins for the management of endometriosis[J]. Fertil Steril,2016, 106(7):1552
[4] Chavarro J E, Rich-Edwards J W, Gaskins A J, et al. Contributions of the nurses’ health studies to reproductive nurses’ health studies to reproductive health research[J]. Am J Public Health，2016,106(9):1669
[5] Mahdian S，Aflatoonian R，Yazdi R S，et al. Macrophage migration inhibitory factor as a potential biomarker of endometriosis[J]. Fertil Steril，2015,103（10）：153
[6] Takebayashi A，Kimura F，Kishi Y，et al. Subpopulations of macrop-hages within eutopic endometrium of endometriosis patients[J]. Am J Reprod Immunol，2015，73（3）：221
[7] Li Y, Adur M K, Kannan A, et al. Progesterone alleviates endometriosis via inhibition of uterine cell proliferation, inflammation and angiogenesis in an immunocompetent mouse model[J]. PLoS One, 2016,11(10):e0165347
[8] Haneya F，Muscalo J J，Weinberg J R． Peritoneal fluid cell populations in infertility patients[J]． Fertil Steril，1981，35(6)：696
[9] Na Y J, Lee D H, Kim S C, et al. Effects of peritoneal fluid from endometriosis patients on the release of monocyte-specific chemokines by leukocytes[J]. Arch Gynecol Obstet, 2011,283(6):1333
[10] Akoum A, Kong J, Metz C, et a1.Spontaneous and stimulated secIelion of monocyte chemotactic protein-l and maerophage migration inhibitory factor by peritoneal macmphages in women with and without endometriosis[J]. Fertil Steril,2002,77(5)：989
[11] Devreotes P，Horwitz A R． Signaling networks that regulate cell migration[J]． Golg Spring Harb Perspeet Biol，2015，7(8)：a005959
[12] DiCosmo-Ponticello C J，Hoover D，Coffman F D，et a1．MIF inhibits monocytic movement through a non-canonical receptor and disruption of temporal Rho GTPase activities in U-937 cells[J]． Cytokine，2014，69(1)：47
[13] 李建，胡玉红，张燕. MIF降低子宫内膜异位症患者腹腔液巨噬细胞的运动速度[J]. 山西医科大学学报，2016，47（4）：373
[14] Beste M T，Pfaffle—Doyle N，Prentice E A，et a1． Molecular network analysis of endometriosis reveals a role for c-Jun-regulated macrophage activation[J]． Sci Transl Med，2014，6(222)：222ral6
[15] Greaves E，Cousins F L, Murray A，et al． A novel mouse model of endometriosis mimics human phenotype and reveals insights into the inflammatory contribution of shed endometrium[J]． Am J Pathol，2014，184(7)：1930
[16] Wu M H，Chuang P C，Lin Y J，et a1． Suppression of annexin A2 by prostaglandin E2 impairs phagocytic ability of peritoneal macrophages in women with endometriosis[J]． Hum Reprod，2013， 28(4)：1045
[17] Kang Y J，Jeung I C，Park A，et a1． An increased level of IL-6 suppresses NK cell activity in peritoneal fluid of patients with endometriosis via regulation of SHP-2expression[J]． Hum Reprod，2014，29(10)：2176
[18] Vercellini P，Viganb P，Somigliana E，et a1． Endometriosis：pathogenesis and treatment[J]． Nat Rev Endecrinol，2014，10(5)：261
[19] Yadav L，Puri N，Rastogi V，et al. Tumour angiogenesis and angiogenic inhibitors: A review [J]. J Clin Diagn Res，2015，9（6）：XE01
[20] Capobianco A, Rovere-Querini P. Endometriosis, a disease of the macrophage[J]. Front Immunol,2013,4(4):9
[21] Na Y J，Yang S H，Baek D W，et al. Effects of peritoneal fluid from endometriosis patients on the release of vascular endothelial growth factor by neutrophils and monocytes [J]. Hum Reprod，2006，21（7）：1846
[22] McLaren J，Prentice A，Charnock-Jones D S，et a1．Vascular endothelial growth factor is produced by peritoneal fluid macrophages in endometriosis and is regulated by ovarian steroids[J]． J Clin Invest，1996，98(2)：482
[23] Lin Y J，Lai M D，Lei H Y，et a1． Neutrophils and macrophages promote angiogenesis in the early stage of endometriosis in a mouse model[J]. Endocrinology, 2006，147(3)：1278
[24] Yang Z，Li L，Chen L，et al. PARP -1 mediates LPS –induced HMGB1 release by macrophages through regulation of HMGB1 acetylation[J]. J Immunol，2014，193（12）：6114
[25] Yang S，Xu L，Yang T，et al. High-mobility group box-1 and its role in angiogenesis[J]. J Leukoc Biol，2014，95（4）：563
[26] Tao Y，zhang Q，Huang W，et al． The peritoneal leptin，MCP-l and TNF-a in the pathogenesis of endometriosis—associated infenility[J]．Am J Repmd Immunol，201l，65(4)：403
[27] Wynn T A, Chawla A, Pollard J W. Macrophage biology in development, homeostasis and disease [J]. Nature, 2013, 496(7446): 445
[28] Wang Y，Chen H，Wang N，et a1．Combined 176-estradiol with TCDD promotes M2 polarization of macrophages in the endometriotic milieu with aid of the interaction between endometrial stromal ceils and macmphages[J]. Plos One，2015，10(5)：eol25559
[29] Bacci M，Capobianco A，Monno A，et al． Macrophages are alternatively activated in patients with endometriosis and required for growth and vascularization of lesions in a mouse model of disease[J]．Am J Pathol，2009，175(2)：547
[30] Yuan M, Li D, An M, et al. Rediscovering peritoneal macrophages in a murine endometriosis model[J]. Hum Reprod, 2017,32(1):94
[31] Xu J, Chi F, Tsukamoto H. Notch signaling and M1 macrophage activation in obesity-alcohol synergism [J]. Clin Res Hepatol Gastroen-terol, 2015, 39 (Suppl 1) : S24
[32] Xu F, Kang Y, Zhang H, et al. Akt1-mediated regulation of macrophage polarization in a murine model of Staphylococcus aureus pulmonary infection [J]. J Infect Dis, 2013, 208(3): 528
[33] 胡玉红，宋美玉. 巨噬细胞活性与子宫内膜异位症发生的关系[J]. 贵阳医学院学报，2016，4（14）：418
[34] 刘黎,胡静平,岳艳.雌激素通过调节IRF4促进巨噬细胞向M2型极化[J].现代免疫学,2013,33(4):285
[35] Greaves E，Temp J，Esnal-Zufiurre A，et a1．Estradiol is a critical mediator of macrophage-nerve cross talk in peritoneal endometriosis[J]． Am J Pathol，2015，185(8)：2286
[36] Zhang X, Zhou M, Guo Y, et al. 1,25-dihydroxyvitamin D(3) promotes high glucose-induced M1 macrophage switching to M2 via the VDR-PPAR gamma signaling pathway[J]. Biomed Res Int, 2015, 2015:157834
[37] Odegaard J I, Ricardo-Gonzalez R R, Goforth M H, et al. Macrophage-specific PPARgamma controls alternative activation and improves insulin resistance [J]. Nature, 2007, 447(7148): 1116
[38] Gordon S，Martinez F O． Alternative activation of macrophages：mechanism andfunctions[J]． Immunity,2010，32(5)：593
[39] Heublein S，Vrekoussis T，Kuhn C，et a1． Inducers of G-protein coupled estrogen receptor(GPER)in endometriosis：potential implications for macrophages and follicle maturation[J]． J Reprod Immunol，2013，97(1)：95
[40] PLies C H，Cannarile M A，Hoves S，et a1． Targeting tumor-associated macrophages with anti-CSF-IR antibody reveals a strategy for cancertherapy[J]． Cancer Ceil，2014，25(6)：846
[41] Zhao Y，Gong P，Chen Y，et a1． Dual suppression of estrogenic and inflammatory activities for targeting of endometriosis[J]． Sci Transl Med，2015，7(271)：27lra9

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[1]刘颂平综述,华克勤审校.巨噬细胞在子宫内膜异位症发生发展中的作用研究现状[J].天津医科大学学报,2018,24(03):271-274.

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