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[1]陈雅婧,王 勇 综述,刘运德,等.C1QBP的多种配体及其在感染和炎症中的研究进展[J].天津医科大学学报,2016,22(01):84-86.
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C1QBP的多种配体及其在感染和炎症中的研究进展(PDF)
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《天津医科大学学报》[ISSN:1006-8147/CN:12-1259/R]

卷:
22卷
期数:
2016年01期
页码:
84-86
栏目:
综述
出版日期:
2016-01-20

文章信息/Info

Title:
-
文章编号:
1006-8147（2016）01-0084-03
作者:
?陈雅婧1; 王 勇2 综述; 刘运德1; 岳 丹1 审校
（1.天津医科大学医学检验学院，天津300203；2. 天津医科大学第二医院泌尿外科，天津市泌尿外科研究所，天津300211）
Author(s):
-
关键词:
C1QBP; 感染; 炎症
Keywords:
-
分类号:
Q7
DOI:
-
文献标志码:
A
摘要:
-
Abstract:
-

参考文献/References:

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[2] Braun L, Ghebrehiwet B, Cossart P. gC1q-R/p32, a C1q-binding protein, is a receptor for the InlB invasion protein of Listeria monocytogenes[J]. EMBO J, 2000. 19(7): 1458

[3] Mahdi F, Madar Z S, Figueroa C D, et al. Factor XII interacts with the multiprotein assembly of urokinase plasminogen activator receptor, gC1qR, and cytokeratin 1 on endothelial cell membranes[J]. Blood, 2002. 99(10): 3585

[4] Matos P, Horn J A, Beards F, et al. A role for the mitochondrial-associated protein p32 in regulation of trophoblast proliferation[J]. Mol Hum Reprod, 2014. 20(8): 745

[5] Xiao K, Wang Y, Chang Z, et al. p32, a novel binding partner of Mcl-1, positively regulates mitochondrial Ca(2+) uptake and apoptosis[J]. Biochem Biophys Res Commun, 2014. 451(2): 322

[6] Kim K B, Yi J S, Nguyen N, et al. Cell-surface receptor for complement component C1q (gC1qR) is a key regulator for lamellipodia formation and cancer metastasis[J]. J Biol Chem, 2011. 286(26): 23093

[7] Ye T, Huang X, Wang X W, et al. Characterization of a gC1qR from the giant freshwater prawn, Macrobrachium rosenbergii[J]. Fish Shellfish Immunol, 2015. 43(1): 200

[8] Ghebrehiwet B, Peerschke E I. Purification of C1q receptors and functional analysis[J]. Methods Mol Biol, 2014. 1100: 319

[9] Westman J, Hansen F C, Olin A I, et al. p33 (gC1q receptor) prevents cell damage by blocking the cytolytic activity of antimicrobial peptides[J]. J Immunol, 2013. 191(11): 5714

[10] Ghebrehiwet B, Jesty J, Vinayagasundaram R, et al. Targeting gC1qR domains for therapy against infection and inflammation[J]. Adv Exp Med Biol, 2013. 735: 97

[11] Castellano G, Woltman A M, Nauta A J, et al. Maturation of dendritic cells abrogates C1q production in vivo and in vitro[J]. Blood, 2004. 103(10): 3813

[12] Hosszu K K, Santiago-Schwarz F, Peerschke E I, et al. Evidence that a C1q/C1qR system regulates monocyte-derived dendritic cell differentiation at the interface of innate and acquired immunity[J]. Innate Immun, 2010. 16(2): 115

[13] Bossi F, Peerschke E I, Ghebrehiwet B, et al. Cross-talk between the complement and the kinin system in vascular permeability[J]. Immunol Lett, 2011. 140(1/2): 7

[14] Kaplan A P. The bradykinin-forming cascade: a historical perspective[J]. Chem Immunol Allergy, 2014. 100: 205

[15] Ghebrehiwet B, Lim B L, Kumar R, et al. gC1q-R/p33, a member of a new class of multifunctional and multicompartmental cellular proteins, is involved in inflammation and infection[J]. Immunol Rev, 2001. 180: 65

[16] Ghebrehiwet B, Jesty J, Xu S, et al. Structure-function studies using deletion mutants identify domains of gC1qR/p33 as potential therapeutic targets for vascular permeability and inflammation[J]. Front Immunol, 2011. 2:58

[17] Ghebrehiwet B, Ji Y, Valentino A, et al. Soluble gC1qR is an autocrine signal that induces B1R expression on endothelial cells[J]. J Immunol, 2014. 192(1): 377

[18] Bossi F, Fischetti F, Regoli D, et al. Novel pathogenic mechanism and therapeutic approaches to angioedema associated with C1 inhibitor deficiency[J]. J Allergy Clin Immunol, 2009. 124(6): 1303

[19] Yang L, Liu X, Liu W, et al. Characterization of complement 1q binding protein of tiger shrimp, Penaeus monodon, and its C1q binding activity[J]. Fish Shellfish Immunol, 2013. 34(1): 82

[20] Sethi S, Herrmann M, Roller J, et al. Blockade of gC1qR/p33, a receptor for C1q, inhibits adherence of Staphylococcus aureus to the microvascular endothelium[J]. Microvasc Res, 2011. 82(1): 66

[21] Shen Y, Naujokas M, Park M, et al. InIB-dependent internalization of Listeria is mediated by the Met receptor tyrosine kinase[J]. Cell, 2000. 103(3): 501

[22] Marino M, Banerjee M, Jonquieres R, et al. GW domains of the Listeria monocytogenes invasion protein InlB are SH3-like and mediate binding to host ligands[J]. EMBO J, 2002. 21(21): 5623

[23] Li X C, Du Z Q, Lan J F, et al. A novel pathogen-binding gC1qR homolog, FcgC1qR, in the Chinese white shrimp, Fenneropenaeus chinensis[J]. Dev Comp Immunol, 2012. 36(2): 400

[24] Fausther-Bovendo H, Vieillard V, Sagan S, et al. HIV gp41 engages gC1qR on CD4+ T cells to induce the expression of an NK ligand through the PIP3/H2O2 pathway[J]. PLoS Pathog, 2010. 6: e1000975

[25] Wang Y, Yang Y, Wu S, et al. p32 is a novel target for viral protein ICP34.5 of herpes simplex virus type 1 and facilitates viral nuclear egress[J]. J Biol Chem, 2014. 289(52): 35795-

[26] Saha P, Ghosh I, Datta K. Increased hyaluronan levels in HABP1/p32/gC1qR overexpressing HepG2 cells inhibit autophagic vacuolation regulating tumor potency[J]. PLoS One, 2014. 9(7): e103208

[27] Peerschke E I, Ghebrehiwet B. cC1qR/CR and gC1qR/p33: observations in cancer[J]. Mol Immunol, 2014. 61(2): 100

[28] Zhang X, Zhang F, Guo L, et al.Interactome analysis reveals that C1QBP (complement component 1, q subcomponent binding protein) is associated with cancer cell chemotaxis and metastasis[J]. Mol Cell Proteomics, 2013. 12(11): 3199

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备注/Memo

备注/Memo:
基金项目 国家自然科学基金青年基金资助项目（81202308和81402094）；天津市应用基础与前沿技术研究计划青年基金项目（14JCQNJC11600）；中国博士后基金面上项目（2013 M541189）；天津医科大学科研基金（2013KYQ04）

作者简介 陈雅婧（1990-），女，硕士在读，研究方向：临床检验诊断学；通信作者：岳丹，E-mail：danyue0705@sina.cn。

更新日期/Last Update: 2016-01-26