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《天津医科大学学报》[ISSN:1006-8147/CN:12-1259/R]

卷:

20卷

期数:

2014年02期

页码:

162-164

栏目:

综述

出版日期:

2014-03-20

文章信息/Info

Title:

-

文章编号:

1006-8147(2014)02-0162-03

作者:

李广斌 综述

（天津市胸科医院病理科，天津 300051）

Author(s):

-

关键词:

肺源性心脏病; 血管舒缩功能; 血管重构; 氧化应激

Keywords:

-

分类号:

R541.5

DOI:

-

文献标志码:

A

摘要:

 

Abstract:

-

参考文献/References:

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[2] Chaouat A. Chronicor pulmonale in COPD[J]. Rev Mal Respir, 2009, 26(10): 1184

[3] Wrobel J P, Thompson B R, Williams T J. Mechanisms of pulmonary hypertension in chronic obstructive pulmonary disease: a pathophysiologic review[J]. Heart Lung Transplant, 2012, 31(6): 557

[4] Heldin C H，Miyazono K，ten Dijke P． TGF-beta signalling from cell membrane to nucleus through SMAD proteins[J]．Nature，2007，390( 6659): 465

[5] Kim S，Yoshiyama M，Izumi Y，et al． Effects of combination of ACE inhibitor and angiotensin receptor blocker on cardiac remodeling，card-iacfunction，and survival in rat heart failure[J]．Circulation，2012，103(1): 48

[6] Fedorova T A, Khimochko T G, Ustinov A A, et al. Angiotensin converting enzyme activity in the clinical course and forming of cor pulmonale in patients with chronic obstructive pulmonary diseases[J]. Klin Med 2013, 8(4): 31

[7] Kobayashi S, Hanagama M, Yanai M, et al. Prevention of chronic obstructive pulmonary disease exacerbation by angi-otensin-converting enzyme inhibitors in individuals with impaired swallowing[J]. Am Geriatr Soc, 2011，59(10):1967

[8] Krushka S I, Butorov I V, Butorov S I, et al. Hartil effects on structural-functional condition of the heart in patients with chronic cor- pulmonale[J]. Tera Che Arkhiv, 2008, 80(9): 63

[9] Butorov I V, Butorov S I, Maksimov V V. Clinical efficiency of course and longterm therapy with the angiotensin-converting enzyme inhibitor perindopril in patients with chronic obstructive lung disease and cor pulmonale[J]. Pro T Bo Leg, 2013，15(6): 20

[10] Shujaat A, Minkin R, Eden E. Pulmonary hypertension and chronic cor pulmo-nale in COPD[J]. Int J COPD 2007, 2(3): 273

[11] Pribylov S A, Konoplia A I, Sirotkin S A. Endothelial dysfunction mediators in patients with chronic cor pulmonale[J]. Klinicheskaia Meditsina, 2006, 84(11): 20

[12] Wick M J, Buesing E J, Wehling C A, et al. Decreased neprilysin and pulmonary vascular remodeling in chronic obstructive pulmonary disease[J]. Am J Respir Crit Care Med，2011, 183(3): 330

[13] 薛明, 陈幸生. 慢性静脉功能不全血管重塑与基质金属蛋白酶关系[J]. 医学综述, 2008, 14(8):1208

[14]Thaktcer V S. Wilson F L, et al. Rote of proteolysis and apoptosisin regre-ssion of pulmonary vascular remodeling[J]. Physic1 Res,2000,49(15):577

[15] Tozzi C A, Christiansen D L, Poiani O J, et al. Excess collagen in hypertensive pulmonary arteries decrease vascular distensibility[J]. Am J Respir Crit Care Ivied, 1994,14(9):13

[16] Vieillard-Baron A, Frisdal E, Eddahibi E, et al. Inhibition of matrix metalloproteinases by lung TIMP-1 gene transfer or doxyeveline aggravates pulmonary hypertention in rats[J].Circulation Research,2000,87(5):418

[17] Ramasamy R, Vannucci S J, Yan S D,et al. Advanced glycationend products and RAGE: a common thread in aging, diabetes neurodegeneration and inflammation[J]. Glycobiology,2009, 15(7):16

[18] Sakata N, Meng J, Takebayashi S. Effects of advanced glycationend products on the proliferation and fibronectin production of smooth muscle cells[J]. Atherscler Thromb, 2010, 7(5): 169

[19] Huang J S, Gu J Y, Chen H C,et al. Role of receptor for advanced glycation end product (RAGE) and the JAK/STAT-signaling pathway in AGE-ind-uced collagen prodction in NRK-49F cells[J]. J Cell Biochem,2010, 8(1): 102

[20] McLennan S V, Martell S K, Yue D K. Effects of mesangium glycation on matrix metallo proteinase activities[J]. Diabetes,2009, 12(5): 2612

[21] Miniati M, Monti S, Basta G, et al. Soluble receptor for advanced glycation end products in COPD: relationship with emphysema and chroni-c cor pulmonale: a case-control study [J]. Res Research ，2011, 12（9）:37

[22] 吕学爱, 关广聚, 晋运玲. 氨基胍对糖尿病大鼠肾脏非酶糖基化的抑制作用[J]. 泰山医学院学报, 2004, 25(3):176

[23] Hsieh T J, Zhang S L, Filep J G, et al. High gluscose stimulates angiotensinogen gene expression via reactive oxygen species generation in rat kidney proximal tubular cells[J].Endocrinology, 2002,143(8):2975

[24] Ogawa S, Mori T, Nako T, et al. Angiotensin Ⅱ Type 1 recepterblockers reduce urinary oxidative stress markers in hypertensive diabetic nephropathy[J]. Hypertension, 2010,47(4):699

[25] Araujo M, Welch W J. Oxidative stress and nitric oxide in kidney function [J].Curr Opin Nephro Hypertens, 2006, 15(1):72

[26] Bohlender J M, Franke S, Stein G, et al. Advanced glycation and products and the kidney[J]. Am Physiol Renal Physiol, 2005, 289(4):645

[27] Fukami K, Ueda S, Yamagishi S, et al. AGEs activate mesangial TGF-β-Smad signaling via an angiotensin Ⅱ type I receptor interaction[J]. Kidney Int, 2004, 66(6):2137

[28] Yuzuru O, Ronald C M, Yutaka Y, et al. Reduction of diabetes-induced oxidative stress,fibrotic cytokine expression,and renal dysfunction inprotein kinase[J]. CB-null mice Diabetes, 2006, 55(11):3112

[29] Bayram E, Atalay C, Kocaturk H, et al. Effects of trimetazidine on lipid peroxidation, antioxidant enzyme activities and plasma brain na-triuretic peptide levels in patients with chronic cor pulmonale [J]. T J Int Med Res， 2005, 33(6): 612

相似文献/References:

备注/Memo

备注/Memo:

 作者简介 李广斌（1975-），男，主治医师，硕士，研究方向：心血管病；E-mail：lgb_2013@163.com。

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更新日期/Last Update: 2014-03-31